By Dr. Jenny Hubball and Dr. Bethany Turner

OSCE


During any examination in an OSCE it is important to understand the pathology and thinking behind each of the signs and symptoms elicited. This applies even if the patient being examined is ‘normal’. This article will explain each of the key findings mentioned in the cardiovascular OSCE examination video, showing you what signs to look out for and what conditions these may indicate.

 

Introduction

Clean your hands (wash them or use alcohol hand rub) before you touch the patient. Introduce yourself to the patient and state your grade, for example third-year medical student, junior doctor or consultant.

Confirm the patient’s details using three points of identification, usually full name, date of birth and NHS or hospital number.

Obtain consent for the examination, and explain what it will involve.

Position the patient appropriately at 45° on the bed.

Exposing the chest may involve exposing the breasts, so offer the patient a chaperone (an impartial observer, usually a health professional) and explain what their role would be. The GMC advises this for intimate examinations, which it says are likely to include examinations of the breasts. Do not assume that the patient does not want one.

Then expose the patient’s chest appropriately (this can be done after examining the patient’s hands, arms, face and neck).

General Inspection

When assessing the colour of the patient, it is important to look for any signs of cyanosis or anaemia. A cyanotic patient would have a bluish tinge to their skin due to a lack of oxygen. A patient with severe anaemia could appear pale. This is because there is less haemoglobin, which usually gives blood (and therefore the skin) its pink colour. It is also important to assess how comfortable and aware (conscious) a patient is. If the patient has an acutely lowered conscious level or is in extreme pain, call for help. 

Hand Examination

When examining the dorsum (back) of the hands, the patient may present with tendinous xanthoma. This is caused by an accumulation of lipids in large foam cells (macrophages filled with fat) within the tendons, resulting in raised, yellowish nodules. This can be caused by familial hyperlipidaemia.

 

Tendinous Xanthoma SimpleMed

Image - Tendinous xanthoma affecting multiple digits of both hands

Creative commons source by Kumar et al. (2008) [CC BY-SA 4.0 (https://creativecommons.org/licenses/by-sa/4.0)]

 

 

When examining the nails, look for signs of splinter haemorrhages and koilonychia. Splinter haemorrhages are thin red or reddish-brown lines of bleeding under the nail, running along its length. They often follow an injury to the nail, but can also be a sign of infective endocarditis (infection of the inner lining of the heart, usually the valves). In endocarditis, a vegetation (a clump of bacteria and clot) on a heart valve can break up into tiny emboli (fragments carried in the blood). These may lodge in the small vessels under the nail.

Koilonychia, also known as spoon nails, is when the nails appear to curve in a concave way. This is most commonly associated with iron deficiency anaemia. It is unclear why this sign occurs.

Splinter Haemorrhage SimpleMed

Image - Splinter haemorrhages under the nails. They can be a sign of infective endocarditis, when tiny emboli lodge in the small vessels under the nails

Public Domain Source Splarka [Public domain]

 

Two skin signs of infective endocarditis are often confused, and tenderness is what separates them. Osler’s nodes are tender, raised nodules, usually on the pulps (pads) of the fingers and toes. Janeway lesions are non-tender, flat, red spots (erythematous macules) on the palms and soles. How both signs form is still debated. Under the microscope, both can show septic emboli (small infected fragments carried in the blood) with microabscesses (tiny collections of pus) in the skin. Remember ‘Osler = Ouch’. Tenderness tells the two apart, and looking only at the palms will miss Osler’s nodes. Neither sign is common: they are found in only about 5 to 15% of people with infective endocarditis.

 

Janeway Lesions SimpleMed

Image - Janeway lesions on the sole of the foot. These are a sign of infective endocarditis, in which septic emboli (small infected fragments) can lodge in the skin

Creative commons source by DeNanneke [CC BY-SA 4.0 (https://creativecommons.org/licenses/by-sa/4.0)]

 

The fingers may show signs of peripheral cyanosis, which would be seen as a bluish tinge to the fingertips. Peripheral cyanosis is due to the inability of the body to perfuse the extremities. This can be caused by low blood pressure, hypothermia (low body temperature) or heart failure.

 

Peripheral Cyanosis SimpleMed

Image - Peripheral cyanosis in the hands. This is due to the fingertips not being adequately perfused

Creative commons source by James Heilman, MD [CC BY-SA 4.0 (https://creativecommons.org/licenses/by-sa/4.0)]

 

Finger clubbing (swelling and rounding of the fingertips) is recognised by several signs. A positive Schamroth’s test (demonstrated in the video) is one of them: the diamond-shaped gap that normally appears between the nails of two opposed fingers is lost. Clubbing is a sign of a variety of different conditions, including heart, lung, liver and bowel disease. In the cardiovascular system, clubbing could be a sign of chronic hypoxia (long-term low oxygen levels), infective endocarditis and some congenital heart defects.

 

Finger Clubbing SimpleMed

Image - The difference in the Schamroth’s window test. The bottom image shows a patient with clubbing as there is no gap between the fingernails

SimpleMed original by Dr. Bethany Turner

 

Capillary refill time is a test used to assess the perfusion of the fingers. This test is carried out by pressing on one of the patient’s fingers for five seconds. Once the finger is released, it should turn from a pale white, back to its normal colour in less than two seconds. If it takes longer than two seconds to reperfuse, then this may be a sign of shock, impaired peripheral perfusion or dehydration.

Pulses

When assessing radial pulse you should look out for its rate and rhythm. Normal pulse rate is around 60-100 bpm (beats per minute).

A collapsing pulse is a strong pulse that drops away suddenly. It reflects a widened pulse pressure (the difference between the systolic and diastolic blood pressure), as in aortic regurgitation (a leaking aortic valve). In the collapsing pulse test, you will feel a ‘tapping’ sensation in the pulse when the arm is raised quickly above the head. Raising the arm makes the sign easier to feel. Before performing this test, it is important to ask if the patient is experiencing any shoulder pain.

To assess the character of the pulse use either the brachial or carotid artery. The pulse's character can be normal, bounding or thready. Other characters include a slow-rising pulse (seen in aortic stenosis, a narrowed aortic valve) and a collapsing pulse (see above).

A bounding pulse, when the pulse feels very strong and throbbing, can be caused by conditions such as aortic regurgitation or severe bradycardia (a slow heart rate). Fever, anaemia and thyrotoxicosis (an overactive thyroid gland) can also cause it. In aortic regurgitation, the pulse pressure is widened leading to a stronger feeling pulse. This is because the left ventricle ejects a larger than normal stroke volume (the volume of blood pumped out with each beat), which tends to raise the systolic pressure. In diastole, however, blood in the aorta runs back into the ventricle through the leaking (incompetent) valve, so the diastolic pressure is low. This results in a large difference between systolic and diastolic pressure, and the pressure changes can be felt as a bounding pulse. Bradycardia allows a longer time for the diastolic run off to occur. This widens the pulse pressure and increases the strength of the pulse.

A thready pulse, one that feels very weak, is seen in conditions when there is a low mean arterial blood pressure (the average pressure in the arteries over one heartbeat), such as in left ventricular failure or hypovolaemia (low blood volume).

Radio-radial delay is when the two radial pulses are not in sync with each other. This can be caused by coarctation of the aorta (narrowing) between the origins of the brachiocephalic trunk (the first branch of the aortic arch, which supplies the right arm and the right side of the head) and the left subclavian artery. This means that blood pumped out of the heart can easily pass through the brachiocephalic trunk. Blood travelling through the left subclavian must pass through the narrowed section. This causes a delay in the blood flow to the left arm. Therefore, the radial pulse in the left arm will be felt slightly after the pulse in the right arm. If the coarctation is after the left subclavian artery, the radial pulses will be felt to be in sync, but the femoral pulse will be delayed. This is called radio-femoral delay.

 

Radio-radial delay may also be caused by an aortic dissection (a tear in the wall of the aorta).

Brachial blood pressure should be monitored to assess for hypo- or hypertension by measuring blood pressure in the upper arm. Read our hypertension guide.

 

Coarctation of the Aorta SimpleMed

Image - Coarctation of the aorta. The coarctation is after the three branches of the aortic arch, so the patient will have radio-femoral delay on examination

SimpleMed original by Dr. Bethany Turner

Facial Examination

On general inspection of the face, assess for mitral facies. This is a sign in which the patient has rosy cheeks with bluish patches. Some authors describe this simply as pinkish-purple patches on the cheeks. It occurs in patients with mitral stenosis (narrowing of the mitral valve) with associated reduced cardiac output.

Patients with hyperlipidaemia may have abnormalities in or around the eyes due to deposition of lipid. Corneal arcus is lipid deposition in the peripheral cornea, seen as a pale ring near the corneal margin. Corneal arcus is common with ageing. Before the age of 45 years, it should prompt assessment for dyslipidaemia (abnormal levels of fats in the blood), in particular familial hypercholesterolaemia. Xanthelasma is similar to tendinous xanthoma in that it is seen when lipid is deposited in the skin around the eyes. It is seen as small raised yellow patches of skin. It does not always mean the blood lipid levels are high: in one study, just over half of the people with xanthelasma had a normal cholesterol level.

 

Xanthelasma SimpleMed

Image - Xanthelasma. There are visible raised yellow patches of skin around the eyes

Creative commons source by Oleg Tymofii [CC BY-SA 4.0 (https://creativecommons.org/licenses/by-sa/4.0)]

 

When a patient pulls down their lower eyelids, look for paleness of the conjunctiva (the thin pink lining of the inner eyelid), which could indicate anaemia.

When assessing the mouth, central cyanosis may be seen, which is a bluish tinge to the mucous membranes. This indicates low oxygen saturation in the blood circulating around the body. Cardiovascular causes include cyanotic congenital heart defects (heart defects present from birth that let blood bypass the lungs) and pulmonary embolism. Do not confuse it with peripheral cyanosis, which is caused by poor circulation rather than poor oxygenation of the blood.

Anaemia from iron or vitamin B12 deficiency can cause glossitis or angular stomatitis. Glossitis (an inflamed tongue) is seen as a glossy, smooth tongue. In iron deficiency this is called atrophic glossitis. In vitamin B12 deficiency the tongue can also be sore and red. Angular stomatitis is seen as dry erythematous (red) changes to the skin at the corners of the mouth.

It is important to assess the state of the teeth (dentition) in a cardiovascular exam, as poor dental hygiene is a common route of entry for the bacteria that can cause infective endocarditis.

 

Angular Stomatitis SimpleMed

Image - Angular stomatitis, which can be a sign of iron deficiency anaemia

Creative commons source by Matthew Ferguson 57 [CC BY-SA 4.0 (https://creativecommons.org/licenses/by-sa/4.0)]

 

Neck Examination

Check the carotid pulses by placing your fingers on the side of the neck, between the sternocleidomastoid muscle (the large neck muscle that runs from behind the ear to the collarbone) and the trachea (windpipe), in the middle third of the neck. Assess the character of the pulse. Do not press high up under the jaw, because the carotid sinus lies there. Pressure on the carotid sinus can slow the heart rate and make the patient faint. Check one side at a time, because pressing on both together can also reduce blood flow to the brain.

Ask the patient to hold their breath and, using the bell of your stethoscope, listen over the carotid arteries for any carotid bruits (abnormal noises). A bruit could indicate narrowing of the carotid arteries, which is a risk factor for stroke. However, listening alone is not reliable: in one study, auscultation missed 44% of people with significant (60% or more) carotid narrowing. A bruit in the neck may also be due to a murmur (an extra sound caused by turbulent blood flow) from aortic stenosis (narrowing of the aortic valve) that radiates to the neck. A good tip is to hold your breath along with the patient. This ensures that the patient does not have to hold their breath for longer than is necessary. 

Assessing the jugular venous pressure (JVP) can give an indication about the pressure in the venous circulation. Pressure can be raised in conditions such as fluid overload or right sided heart failure. To perform the test, make sure the patient is sitting at 45° with their head turned to the left. Identify the internal jugular vein which usually lies between the two heads of the sternocleidomastoid muscle as it inserts into the sternum and clavicle.If a pulse is visible, measure the height between the sternal angle and the border of the pulsation to assess whether the pressure is raised.

 

Jugular Venous Pressure SimpleMed

Image - How to measure the JVP. Angle the patient at 45° and ask them to turn their head away from you and look for a pulsation in their neck. A pulsation more than 3 cm above the sternal angle suggests pathology such as fluid overload or heart failure

SimpleMed original by Dr. Bethany Turner

Chest Inspection

Before examining the chest, it is important to allow time for a general inspection. Look for abnormalities such as pectus excavatum. In this condition, the sternum is ‘sunken’ in the chest wall, giving a caved-in appearance. Also look for pectus carinatum, also called ‘pigeon chest’. Here the sternum is ‘pushed out’ of the chest wall, giving a pointed appearance. These congenital abnormalities can affect breathing.

Look for any signs of trauma, any asymmetry, any surgical scars, indwelling pacemakers (devices under the skin that keep the heartbeat regular) and any visible pulsation.

Chest Palpation

Before palpating the chest make sure the patient isn’t in any pain.

Place the heel of the hand over the left edge of the sternum and check for a parasternal heave. This can be felt as a pushing sensation on your hand and suggests right ventricular hypertrophy (a thickened right ventricle). A parasternal heave can also occur in tricuspid regurgitation and mitral regurgitation (backward leaks through those valves).

Feel in the left fifth intercostal space (the gap between two ribs) in the mid-clavicular line (a vertical line down from the middle of the collarbone) for the apex beat (the lowest and outermost point where you can feel the heartbeat). If it is displaced laterally, this suggests left ventricular dilatation (enlargement of the left ventricle, or cardiomegaly), or a mediastinal shift (the central chest contents pushed to one side) from a large pleural effusion (fluid around the lung) or pneumothorax (air around the lung). Left ventricular hypertrophy classically gives a sustained, heaving apex beat that is not displaced.

Place your hand over each valve area and feel for any heaves or thrills (felt like vibrations under the hand). A thrill indicates turbulent flow around a valve due to regurgitation (a backward leak through the valve) or stenosis (narrowing of the valve). Thrills are only felt with loud murmurs.

Valve areas (the places on the chest wall where each valve is best heard):

  • Aortic: right sternal edge, second intercostal space
  • Pulmonary: left sternal edge, second intercostal space
  • Tricuspid: left sternal edge, fifth intercostal space
  • Mitral: mid-clavicular line, fifth intercostal space

 

Heart Valve Locations SimpleMed

Image - The four valve areas where the stethoscope should be placed to listen to the valves.
A: Aortic, P: Pulmonary, T: Tricuspid, M: Mitral

SimpleMed original

 

Chest Auscultation

Listen with a stethoscope over the valve areas for any murmurs which could indicate turbulent flow caused by stenosis or regurgitation of the valves.

Aortic valve stenosis murmurs can radiate to the neck so listen upwards towards the neck to check for this.

There are two common accentuation manoeuvres, which are positions that help you hear certain murmurs more clearly:

  • Sit the patient forward and listen with the diaphragm at the left sternal edge (Erb’s point, third to fourth intercostal space). Listen in held expiration (with the breath held out) for aortic regurgitation. Listening only over the aortic area is a common way to miss the murmur.
  • Roll the patient onto their left side and listen over the mitral area while the patient breathes out, for mitral stenosis or regurgitation. Use the bell of the stethoscope for mitral stenosis. Move the stethoscope towards the axilla (armpit) to listen for radiation of mitral valve murmurs.

 

Heart Valve Auscultation Sounds SimpleMed

Image - The different sounds that can be heard when auscultating heart valves

SimpleMed original by Dr. Bethany Turner

Back Examination

Auscultate the lung bases to listen for any crackles (bubbling sounds) which could indicate pulmonary oedema due to left sided heart failure.

Press firmly on the skin in the sacral area for a few seconds to check for any sacral oedema which could suggest fluid overload or heart failure.

 

Sacral Oedema SimpleMed

Image - How to check for sacral oedema

SimpleMed original

Examination of the Feet

Press firmly on the skin around the ankles for a few seconds to check for any pitting oedema (swelling that leaves a dent when pressed) which could suggest fluid overload or heart failure.

Check the pedal pulses in both feet. To feel for the posterior tibial pulse, press your fingers behind the medial malleolus (the bony bump on the inner side of the ankle). To feel for the dorsalis pedis pulse (on the top of the foot), press your fingers lateral to (on the outer side of) the extensor hallucis longus tendon (the tendon that lifts the big toe). Absence of these pulses may indicate peripheral arterial disease (PAD, also called peripheral vascular disease), where narrowed leg arteries restrict the blood supply to the legs.

 

Dorsalis Pedis Pulse SimpleMed

Posterior Tibial Pulse SimpleMed

Image - How to check the pedal pulses. The dorsalis pedis pulse is felt on the top of the feet lateral to the extensor hallucis longus tendon. The posterior tibial pulse is felt behind the medial malleolus

Both images are SimpleMed originals

 

Completing the Examination

Thank the patient and wash your hands.

Summarise your findings.

To complete the examination, suggest performing a 12-lead ECG to look for any arrhythmias (abnormal heart rhythms). Suggest an examination of the peripheral vasculature (the pulses and blood vessels of the limbs). Suggest a urine dip to check for proteinuria (protein in the urine) or haematuria (blood in the urine). Suggest relevant blood tests, for example high-sensitivity troponin (a marker of heart muscle damage) if a heart attack is suspected, and NT-proBNP (N-terminal pro-B-type natriuretic peptide, a marker used when heart failure is suspected).

Edited by: Dr. Ben Appleby

Reviewed by: Dr. Thomas Burnell and Dr. Marcus Judge

 

Quiz

Preview the Cardiovascular System OSCE Examination quiz